C. elegans Kallmann syndrome protein KAL-1 interacts with syndecan and glypican to regulate neuronal cell migrations

Article Abstract:

The role of Caenorhabditis elegans Kallmann syndrome protein KAL-1, a member of anosmin-1 protein family, in promoting neuroblast migration is analyzed. KAL-1 interacts with heparan sulfate proteoglycans to cause neuronal cell movements.

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Multiple redundant Wnt signaling components function in two processes during C. elegans vulval development

Article Abstract:

The role of Wnt signaling pathways, a complex network of proteins in the development of vulva in Caenorhabditis elegans is studied.

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N-ethylmaleimide sensitive factor is required for fusion of the C. elegans uterine anchor cell

Article Abstract:

Biochemical processes in animals and the role of N-ethymaleimide in this process is examined.

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